Seborrheic Dermatitis
Seborrhoeic dermatitis is a chronic reaction to Malassezia yeast in oil-rich areas, causing flaking and redness across the scalp, brows and the folds of the face.
Clinic and treatment images
Tap to enlargeDo you recognise these signs?
If one or more of these applies to you, we recommend a dermatological assessment.
- Greasy, yellowish flakes on the scalp over an itchy, reddened base
- Scaly red patches between the brows, along the nose and around the ears
- Oily flaking with mild itch over the mid-chest and the beard area
- A waxing-and-waning course that shifts with season, stress and fatigue
Causes and triggers
- An exaggerated inflammatory response to Malassezia yeast native to the skin
- Increased sebum output and an oil-rich skin surface that feeds the yeast
- Cold, dry weather, stress and disrupted sleep acting as triggers
- A link with immune-suppressing conditions and certain neurological disorders
How we treat it
Antifungal-led care: yeast-reducing shampoos and creams to rein in inflammation
Calcineurin inhibitors for cortisone-free control alongside gentle barrier repair
A cleansing routine that spares the oil balance and avoids harsh irritant products
For stubborn disease, a systemic review of zinc, vitamin D and gut health
Why Clinovia?
Psoriasis and eczema are not only a surface problem. We assess the skin barrier, inflammation, nutrition and lifestyle, comorbidities and dermatological treatment together. Evidence-based dermatology remains the backbone; nutrition and lifestyle support that plan.
The Gut–Skin Relationship
We review digestive symptoms, dietary pattern and food-sensitivity history. The link between gut microbiome and inflammatory skin disease is still being researched, so it is addressed alongside — never instead of — dermatological treatment.
Cortisone-Free Protocol
First-line calcineurin and PDE-4 inhibitors, ceramide-based barrier repair and phototherapy instead of steroids; systemic and biologic steps when needed.
Systemic Repair
Vitamin D, zinc, omega-3 and iron levels plus thyroid and insulin resistance are screened, and any deficits found are built into the plan.
Measurable Follow-up
PASI/EASI and DLQI scores are tracked alongside laboratory values; steps that do not deliver a response are removed from the plan.
Dandruff is the mildest end of seborrhoeic dermatitis: flaking only, without marked redness or inflammation. Seborrhoeic dermatitis adds redness, itch and spread. Because Malassezia yeast underlies both, antifungal-led care helps in either case, adjusted to how active the skin is.
Seborrhoeic dermatitis is not contagious and has nothing to do with poor hygiene. It follows a chronic, relapsing course, so we cannot promise it will vanish for good. With antifungal care and trigger management, however, most people reach long, comfortable spells between flares.
Yes. For infant eczema and paediatric psoriasis we use age-appropriate topical stepping, a moisturising routine and, when indicated, paediatric phototherapy dosing.
Our standard protocols are cortisone-free. We work with topical calcineurin inhibitors, PDE-4 inhibitors, ceramide-based barrier repair, NB-UVB phototherapy and biologic agents when needed. Cortisone is considered only for very short, unavoidable situations and with the patient's consent.
Other conditions
Atopic DermatitisAtopic dermatitis is a chronic form of eczema in which a weakened skin barrier drives itch and redness; it runs in flares and remissions and often settles in the body's flexures.
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Contact EczemaContact eczema is inflammation triggered when a substance touching the skin acts as an allergen or irritant; it appears as sharply bordered redness and itch at the site of contact.
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Hand & Foot EczemaHand and foot eczema brings dryness, itch and cracking to the palms, fingers and soles; it recurs and worsens with wet work and damp, sweaty footwear.
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NB-UVB PhototherapyNarrowband UVB at 311 nm calms inflammation in psoriasis and eczema without cortisone. Sessions run two to three times a week, with a typical course of 20–30 and PASI/EASI tracking.
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Biologic TreatmentsBiologic drugs selectively block targets such as IL-17 and IL-23 in psoriasis and IL-4/13 in eczema. They are considered in moderate-to-severe, resistant disease after screening.
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Cortisone-Free Topical PlanA cortisone-free topical plan combines calcineurin inhibitors, PDE-4 inhibitors and ceramide barrier repair to control inflammation without steroid side effects.
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Let's build the right plan for your skin today
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- Same-day callback
- Consultation in five languages
- Written treatment plan
- Defterdar, Fethi Çelebi Cd. No:15, 34050 Eyüpsultan/İstanbul, Türkiye
- Mon–Sat: 09:00 – 19:00